DR5/TNFRSF10B/CD262/TRAIL-R2 Drug Discovery Landscape & Assay Solutions

Market Intelligence, Clinical Progress, and High-Purity Reagents for TRAIL-Receptor Targeted Cancer Therapy Development.

Key Molecular Characteristics

  • Death Domain: Contains a functional death domain (UniProt O14763 domain), essential for DISC formation and downstream caspase activation.
  • Key Mutations: Documented single-nucleotide polymorphisms in dbSNP: rs1129424 (VAR_016153), rs1047266 (VAR_016154), and rs13265018 (VAR_059831). These variations may affect receptor function and therapeutic response.
  • Alternative Names: Also known as TNFRSF10B, DR5, CD262, and TRAIL-R2. Primary UniProt entry: O14763.

TarMart Solution Ecosystem & Related Targets

Comprehensive reagent toolkit for DR5/TNFRSF10B drug discovery.

Component / Network Product Description Product Link
Antigen TNFRSF10B (DR5) ECD-Fc Fusion Protein
High purity (>95%), Endotoxin <1EU/µg, Sequence Verified. HEK293 expressed (native glycosylation). Contains complete cysteine-rich domains (CRD1-3).
View TNFRSF10B Products
Gene Delivery TNFRSF10B Promise-ORF / Lentivirus Premade Particles
Full-length ORF with native signal peptide for stable cell line construction. Preserves death domain (DD) signaling integrity.
View TNFRSF10B Products
Benchmark Ab Anti-DR5 (Reference Sequences)
Recombinant agonistic antibody controls (monovalent/bivalent formats available). Sequence Verified.
View TNFRSF10B Products
Validator TNFRSF10B siRNA Set (3 Unique Sequences)
For knockdown verification and apoptosis rescue assays.
View TNFRSF10B Products
Related Target: DR4 TNFRSF10A (TRAIL-R1/DR4)
Pro-apoptotic paralog; critical for selectivity screening and off-target analysis.
View TNFRSF10A Products
Related Target: DcR1 TNFRSF10C (TRID)
Decoy receptor (GPI-anchored, no death domain); major pathway competitor for TRAIL binding.
View TNFRSF10C Products
Related Target: DcR2 TNFRSF10D (TRUNDD)
Decoy receptor (truncated death domain); screening essential for agonist specificity.
View TNFRSF10D Products
Related Target: FAP Fibroblast Activation Protein
TME-restricted bispecific DR5 clustering target for tumor-localized activation.
View FAP Products
Critical Assay Challenge The TarMart Advantage (Technical Spec)
Oligomerization-Dependent Activity (Clustering Requirement) Native-conformation ECD-Fc preserves CRD domain architecture; supports tetravalent/hexavalent agonist binding studies and clustering assays.
Cross-species Cyno/Mouse Evaluation Human/Cyno/Mouse TNFRSF10B ortholog proteins available with >95% purity; critical for preclinical toxicology bridging studies.
Selectivity vs. Death Receptor 4 (DR4) & Decoys Homolog panel (TNFRSF10A, TNFRSF10C, TNFRSF10D) strictly verified by mass spec; enables counter-screening for true DR5-selective agonists.
Internalization Efficiency (ADC Development) Lentivirus-based stable overexpression lines in HEK293 and tumor backgrounds; preserves endosomal trafficking for ADC payload delivery validation.
Lack of Functional Controls Clinical benchmark antibody sequences (biosimilar) and validated siRNA included for assay specificity verification.

Global Clinical Landscape & Future Outlook

The DR5 (TNFRSF10B) therapeutic landscape has evolved significantly following the failure of first-generation bivalent antibodies (e.g., drozitumab, conatumumab). Early bivalent antibodies failed to induce sufficient receptor clustering and suffered from hepatotoxicity due to systemic FcγR-mediated crosslinking. Current R&D focuses on multivalent agonist formats (tetravalent/hexavalent/IgM) capable of inducing the high-order clustering required for DISC formation and caspase-8 activation.

Key clinical-stage assets include:

  • INBRX-109 (Inhibrx): Tetravalent DR5 agonist, Phase 1/2 in sarcoma.
  • APG-880 (Apogenix): Hexavalent COMR®-based construct, targeting colorectal and gastric cancers.
  • IGM-8444 (IGM Biosciences): IgM-based decavalent agonist, in solid tumors.
  • APX025 (Apexigen): Bispecific DR5 x tumor antigen.

The next wave also includes ADC strategies leveraging DR5’s tumor-selective expression and internalization capacity, as well as TME-restricted bispecifics (e.g., FAP x DR5) to avoid systemic liver toxicity. Combination therapies with chemotherapy, Bcl-2 inhibitors, or checkpoint inhibitors are expected to become standard clinical development paths.

Competitive Modality & Indication Snapshot

Modality Representative Players Key Indications Critical Assay Need (Why TarMart?)
Multivalent / IgM IGM Biosciences, Genmab Solid Tumors, Colorectal Cancer Clustering Assay (Need high-purity ECD-Fc & Stable Cell Lines)
Tetravalent Agonist Inhibrx (INBRX-109) Sarcoma, Solid Tumors Cell Death Assay (Need Lentivirus stable lines for functional clustering validation)
Hexavalent Agonist Apogenix (APG-880) Colorectal, Gastric Oligomerization Assay (Need native-conformation ECD-Fc for avidity studies)
Bispecific (TME-targeted) Biotheus, Roche, Apexigen Gastric, Pancreatic Selectivity Panel (Need TNFRSF10A/C/D homologs for off-target screening)
Antibody-Drug Conjugates (ADC) Various early-stage, Daiichi Sankyo (Legacy) DR5-high Solid Tumors Internalization Assay (Need high-purity ECD-Fc for binding kinetics + Lentivirus lines for trafficking)

Live DR5/TNFRSF10B R&D Tracker

Market data changes daily. Access the latest global pipeline status directly: